Using Proteomic Analysis & MEMRI to Understand Axonal Transport Deficits and Improvements in a Mouse Model of Alzheimers Disease

Keywords

ability acidic activated addition additional aggregated allow animals antioxidant assessed basis begin begun better biology biophysics blot boosted brain characterized cognitive college colored combined commercialized conferred conjunction continuing contributes contributing control decline decrease deficits degradation deposition detecting determined development differential differentially disciplinary disease disrupts domain domains dyes dynamics electrophoresis example excess exhibited exist expressed expression extracts flow fluorescent fold free functions gels genetically gets green hallmark homogenates identified identify identity implicate improvements indicating inflammation insights intracellular involved known labeled least loaded long macrophage marked mass mechanism mechanisms medicine mice model models molecular mouse natl normalizing organization overlap oxidative parts pathologies pathology pave physiology plaques potentially precursor prepared presence prevents previous process progressive promotes protection protein proteins quench radicals recoveries recovery recruitment reduces reducing relevant response sample samples senile separated series software spec specifically spots stimulating strategy stress structural studies tandem tangles third tissue transport treatment understand understanding upon validating ward western