Lack of Extracellular Adenosine Formation Promotes Lipolysis, Intromyocellular Lipid Deposition & Peripheral Insulin Resistance

Keywords

abbreviations abdominal accompanied accumulation acid acids acquisition additional adult aimed almost alterations altered animals anterior arbitrary bandwidth blood body breakdown cardiology carried cells characterize characterized chemical choline chow classification clearly coil compensation composition conditions consequences contamination content contents controls critically decrease deep deficient deposition derived development diet diseases displayed disrupted distribution eddy enhanced entire equipped events experimental extent extracellular extracellularly fatty final findings formation formed fraction free glucose glutamate gradient hepatic impaired important in vivo insulin integral intra involved known lack length like lipid lipids liver lobe localization localized located male many measured mediated metabolic mice micro minor modulator molecular months muscle mutant outer particular peripheral polyunsaturated predominantly press promotes proton quantification receive receptors recorded reduce reduced related release resistance revealed sampling selective serum shielded significantly similarly spectra step strength subcutaneous suggest superficial suppressed suppression surface syndrome tibia transmit triggered triglycerides ubiquitous unchanged unit units unveiled vapor vertical visceral volume water whether wide